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cholate d4  (steraloids inc)


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    Structured Review

    steraloids inc cholate d4
    Figure 2. Histological features of ANIT-induced cholestasis model. (A) H&E and Sirius red stained liver sections from normal group, 8w model group, 12w model group and YCHT-treated group. Normal, healthy rats; 8w Model, ANIT treatment for 8 weeks; 12w Model, ANIT treatment for <t>12</t> weeks; YCHT-treated, ANIT- induced cholestasis rats treated with YCHT from 9th week to 12th week. Original magnifications × 100 for H&E, and × 200 for Sirius red. (B) Immunofluorescence analysis of CK19 in liver sections. (C) The positive area of CK19 in different groups. (D) Immunofluorescence analysis of Collagen I in liver sections. (E) The positive area of Collagen I in different groups. *P < 0.05; **P < 0.01; ***P < 0.001.
    Cholate D4, supplied by steraloids inc, used in various techniques. Bioz Stars score: 93/100, based on 5 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/cholate d4/product/steraloids inc
    Average 93 stars, based on 5 article reviews
    cholate d4 - by Bioz Stars, 2026-05
    93/100 stars

    Images

    1) Product Images from "Herbal medicine Yinchenhaotang protects against α-naphthylisothiocyanate-induced cholestasis in rats."

    Article Title: Herbal medicine Yinchenhaotang protects against α-naphthylisothiocyanate-induced cholestasis in rats.

    Journal: Scientific reports

    doi: 10.1038/s41598-017-04536-5

    Figure 2. Histological features of ANIT-induced cholestasis model. (A) H&E and Sirius red stained liver sections from normal group, 8w model group, 12w model group and YCHT-treated group. Normal, healthy rats; 8w Model, ANIT treatment for 8 weeks; 12w Model, ANIT treatment for 12 weeks; YCHT-treated, ANIT- induced cholestasis rats treated with YCHT from 9th week to 12th week. Original magnifications × 100 for H&E, and × 200 for Sirius red. (B) Immunofluorescence analysis of CK19 in liver sections. (C) The positive area of CK19 in different groups. (D) Immunofluorescence analysis of Collagen I in liver sections. (E) The positive area of Collagen I in different groups. *P < 0.05; **P < 0.01; ***P < 0.001.
    Figure Legend Snippet: Figure 2. Histological features of ANIT-induced cholestasis model. (A) H&E and Sirius red stained liver sections from normal group, 8w model group, 12w model group and YCHT-treated group. Normal, healthy rats; 8w Model, ANIT treatment for 8 weeks; 12w Model, ANIT treatment for 12 weeks; YCHT-treated, ANIT- induced cholestasis rats treated with YCHT from 9th week to 12th week. Original magnifications × 100 for H&E, and × 200 for Sirius red. (B) Immunofluorescence analysis of CK19 in liver sections. (C) The positive area of CK19 in different groups. (D) Immunofluorescence analysis of Collagen I in liver sections. (E) The positive area of Collagen I in different groups. *P < 0.05; **P < 0.01; ***P < 0.001.

    Techniques Used: Staining, Immunofluorescence

    Figure 3. mRNA expression of IL-6, IL-10, TGF-β1, IL-17A, IL-17F, and α-SMA in the liver. Normal, healthy rats; 8w Model, ANIT treatment for 8 weeks; 12w Model, ANIT treatment for 12 weeks; Yinchenhaotang- treated, ANIT-induced cholestasis rats treated with Yinchenhaotang from 9th week to 12th week. All data are presented as mean ± SEM. (n = 8–12; *P < 0.05; **P < 0.01).
    Figure Legend Snippet: Figure 3. mRNA expression of IL-6, IL-10, TGF-β1, IL-17A, IL-17F, and α-SMA in the liver. Normal, healthy rats; 8w Model, ANIT treatment for 8 weeks; 12w Model, ANIT treatment for 12 weeks; Yinchenhaotang- treated, ANIT-induced cholestasis rats treated with Yinchenhaotang from 9th week to 12th week. All data are presented as mean ± SEM. (n = 8–12; *P < 0.05; **P < 0.01).

    Techniques Used: Expressing

    Figure 5. The mRNA expression of liver FXR, CAR, VDR, TGR5, NTCP, BSEP, OATP1a1, MRP2, MRP3, and MRP4, and ileum ASBT. Normal, healthy rats; Model, ANIT treatment for 12 weeks; YCHT, ANIT-induced cholestasis rats treated with Yinchenhaotang from 9th week to 12th week. All data are presented as mean ± SEM. (n = 8–12; *P < 0.05; **P < 0.01).
    Figure Legend Snippet: Figure 5. The mRNA expression of liver FXR, CAR, VDR, TGR5, NTCP, BSEP, OATP1a1, MRP2, MRP3, and MRP4, and ileum ASBT. Normal, healthy rats; Model, ANIT treatment for 12 weeks; YCHT, ANIT-induced cholestasis rats treated with Yinchenhaotang from 9th week to 12th week. All data are presented as mean ± SEM. (n = 8–12; *P < 0.05; **P < 0.01).

    Techniques Used: Expressing



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    Figure 2. Histological features of ANIT-induced cholestasis model. (A) H&E and Sirius red stained liver sections from normal group, 8w model group, 12w model group and YCHT-treated group. Normal, healthy rats; 8w Model, ANIT treatment for 8 weeks; 12w Model, ANIT treatment for <t>12</t> weeks; YCHT-treated, ANIT- induced cholestasis rats treated with YCHT from 9th week to 12th week. Original magnifications × 100 for H&E, and × 200 for Sirius red. (B) Immunofluorescence analysis of CK19 in liver sections. (C) The positive area of CK19 in different groups. (D) Immunofluorescence analysis of Collagen I in liver sections. (E) The positive area of Collagen I in different groups. *P < 0.05; **P < 0.01; ***P < 0.001.
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    Image Search Results


    Figure 2. Histological features of ANIT-induced cholestasis model. (A) H&E and Sirius red stained liver sections from normal group, 8w model group, 12w model group and YCHT-treated group. Normal, healthy rats; 8w Model, ANIT treatment for 8 weeks; 12w Model, ANIT treatment for 12 weeks; YCHT-treated, ANIT- induced cholestasis rats treated with YCHT from 9th week to 12th week. Original magnifications × 100 for H&E, and × 200 for Sirius red. (B) Immunofluorescence analysis of CK19 in liver sections. (C) The positive area of CK19 in different groups. (D) Immunofluorescence analysis of Collagen I in liver sections. (E) The positive area of Collagen I in different groups. *P < 0.05; **P < 0.01; ***P < 0.001.

    Journal: Scientific reports

    Article Title: Herbal medicine Yinchenhaotang protects against α-naphthylisothiocyanate-induced cholestasis in rats.

    doi: 10.1038/s41598-017-04536-5

    Figure Lengend Snippet: Figure 2. Histological features of ANIT-induced cholestasis model. (A) H&E and Sirius red stained liver sections from normal group, 8w model group, 12w model group and YCHT-treated group. Normal, healthy rats; 8w Model, ANIT treatment for 8 weeks; 12w Model, ANIT treatment for 12 weeks; YCHT-treated, ANIT- induced cholestasis rats treated with YCHT from 9th week to 12th week. Original magnifications × 100 for H&E, and × 200 for Sirius red. (B) Immunofluorescence analysis of CK19 in liver sections. (C) The positive area of CK19 in different groups. (D) Immunofluorescence analysis of Collagen I in liver sections. (E) The positive area of Collagen I in different groups. *P < 0.05; **P < 0.01; ***P < 0.001.

    Article Snippet: 9Scientific RepoRts | 7: 4211 | DOI:10.1038/s41598-017-04536-5 taurodeoxycholate (TDCA), glycoursodeoxycholate (GUDCA), glycohyodeoxycholate (GHDCA), glycochenodeoxycholate (GCDCA), glycodeoxycholate (GDCA), cholate-d4 (5β-Cholanic acid-3α, 7α, 12α-triol-2,2,4,4-d4, CA-d4), glycocholate-d4 (5β-Cholanic acid-3α, 7α, 12α-triol N-(Carboxymethyl)-amide-2,2,4,4-d4, GCA-d4), lithocholate-d4 (5β-Cholanic acid-3α-ol-2,2,4,4-d4, LCA-d4), and deoxycholate-d4 (5β-Cholanic acid-3α, 12α-diol-2,2,4,4-d4, DCA-d4) were purchased from Steraloids Inc. (Newport, RI).

    Techniques: Staining, Immunofluorescence

    Figure 3. mRNA expression of IL-6, IL-10, TGF-β1, IL-17A, IL-17F, and α-SMA in the liver. Normal, healthy rats; 8w Model, ANIT treatment for 8 weeks; 12w Model, ANIT treatment for 12 weeks; Yinchenhaotang- treated, ANIT-induced cholestasis rats treated with Yinchenhaotang from 9th week to 12th week. All data are presented as mean ± SEM. (n = 8–12; *P < 0.05; **P < 0.01).

    Journal: Scientific reports

    Article Title: Herbal medicine Yinchenhaotang protects against α-naphthylisothiocyanate-induced cholestasis in rats.

    doi: 10.1038/s41598-017-04536-5

    Figure Lengend Snippet: Figure 3. mRNA expression of IL-6, IL-10, TGF-β1, IL-17A, IL-17F, and α-SMA in the liver. Normal, healthy rats; 8w Model, ANIT treatment for 8 weeks; 12w Model, ANIT treatment for 12 weeks; Yinchenhaotang- treated, ANIT-induced cholestasis rats treated with Yinchenhaotang from 9th week to 12th week. All data are presented as mean ± SEM. (n = 8–12; *P < 0.05; **P < 0.01).

    Article Snippet: 9Scientific RepoRts | 7: 4211 | DOI:10.1038/s41598-017-04536-5 taurodeoxycholate (TDCA), glycoursodeoxycholate (GUDCA), glycohyodeoxycholate (GHDCA), glycochenodeoxycholate (GCDCA), glycodeoxycholate (GDCA), cholate-d4 (5β-Cholanic acid-3α, 7α, 12α-triol-2,2,4,4-d4, CA-d4), glycocholate-d4 (5β-Cholanic acid-3α, 7α, 12α-triol N-(Carboxymethyl)-amide-2,2,4,4-d4, GCA-d4), lithocholate-d4 (5β-Cholanic acid-3α-ol-2,2,4,4-d4, LCA-d4), and deoxycholate-d4 (5β-Cholanic acid-3α, 12α-diol-2,2,4,4-d4, DCA-d4) were purchased from Steraloids Inc. (Newport, RI).

    Techniques: Expressing

    Figure 5. The mRNA expression of liver FXR, CAR, VDR, TGR5, NTCP, BSEP, OATP1a1, MRP2, MRP3, and MRP4, and ileum ASBT. Normal, healthy rats; Model, ANIT treatment for 12 weeks; YCHT, ANIT-induced cholestasis rats treated with Yinchenhaotang from 9th week to 12th week. All data are presented as mean ± SEM. (n = 8–12; *P < 0.05; **P < 0.01).

    Journal: Scientific reports

    Article Title: Herbal medicine Yinchenhaotang protects against α-naphthylisothiocyanate-induced cholestasis in rats.

    doi: 10.1038/s41598-017-04536-5

    Figure Lengend Snippet: Figure 5. The mRNA expression of liver FXR, CAR, VDR, TGR5, NTCP, BSEP, OATP1a1, MRP2, MRP3, and MRP4, and ileum ASBT. Normal, healthy rats; Model, ANIT treatment for 12 weeks; YCHT, ANIT-induced cholestasis rats treated with Yinchenhaotang from 9th week to 12th week. All data are presented as mean ± SEM. (n = 8–12; *P < 0.05; **P < 0.01).

    Article Snippet: 9Scientific RepoRts | 7: 4211 | DOI:10.1038/s41598-017-04536-5 taurodeoxycholate (TDCA), glycoursodeoxycholate (GUDCA), glycohyodeoxycholate (GHDCA), glycochenodeoxycholate (GCDCA), glycodeoxycholate (GDCA), cholate-d4 (5β-Cholanic acid-3α, 7α, 12α-triol-2,2,4,4-d4, CA-d4), glycocholate-d4 (5β-Cholanic acid-3α, 7α, 12α-triol N-(Carboxymethyl)-amide-2,2,4,4-d4, GCA-d4), lithocholate-d4 (5β-Cholanic acid-3α-ol-2,2,4,4-d4, LCA-d4), and deoxycholate-d4 (5β-Cholanic acid-3α, 12α-diol-2,2,4,4-d4, DCA-d4) were purchased from Steraloids Inc. (Newport, RI).

    Techniques: Expressing

    Bile acid profiles in the GI tract (duodenum, ileum, cecum, and rectum) of control rats

    Journal: The FASEB Journal

    Article Title: Alteration of bile acid metabolism in the rat induced by chronic ethanol consumption

    doi: 10.1096/fj.13-231860

    Figure Lengend Snippet: Bile acid profiles in the GI tract (duodenum, ileum, cecum, and rectum) of control rats

    Article Snippet: Chemicals and reagents Lithocholate (LCA), nordeoxycholate (NDCA), murideoxycholate (MDCA), hyodeoxycholate (HDCA), chenodeoxycholate (CDCA), deoxycholate (DCA), dehydrocholate (DHCA), glycocholate (GCA), taurolithocholate (TLCA), glycolithocholate (GLCA), α-muricholate (α-MCA), β-muricholate (β-MCA), ω-muricholate (ω-MCA), λ-muricholate (λ-MCA), cholate (CA), 7-dehydrocholate (7-DCA), methyl deoxycholate (methyl DCA), 6,7-diketodeoxycholate (6,7-DKDCA), tauro α-muricholate (T α-MCA), tauro β-muricholate (T β-MCA), taurocholate (TCA), tauroursodeoxycholate (TUDCA), taurohyodeoxycholate (THDCA), taurochenodeoxycholate (TCDCA), taurodeoxycholate (TDCA), glycoursodeoxycholate (GUDCA), glycohyodeoxycholate (GHDCA), glycochenodeoxycholate (GCDCA), glycodeoxycholate (GDCA), cholate-d4 (CA-d4; 5β-cholanic acid-3α,7α,12α-triol-2,2,4,4-d4), glycocholate-d4 (GCA-d4; 5β-cholanic acid-3α,7α,12α-triol N -(carboxymethyl)-amide-2,2,4,4-d4), lithocholate-d4 (LCA-d4; 5β-cholanic acid-3α-ol-2,2,4,4-d4), and deoxycholate-d4 (DCA-d4; 5β-cholanic acid-3α, 12α-diol-2,2,4,4-d4) were purchased from Steraloids, Inc. (Newport, RI, USA).

    Techniques: Control